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All currently available inotropic agents act to increase Ca2+ for activation in both normal and failing myocardium (Hurst). The use of inotropic agents in the treatment of CHF is predicated on the finding that a major contributing factor in reducing ventricular performance results from depression of myocardial contractility and that this can be reversed, or at least improved, by inotropic drugs.
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why are proteins so versatile?
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