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In infant botulism, viable botulinal spores are ingested and upon germination in the intestinal tract, toxin is synthesized. It is confined to infants under a year of age. High number of spores is found in the faeces of infants during the acute phase of the disease. It appears that ingested endospores, which maybe present in honey or other baby foods, germinate in the infant's intestine. C. botulinum then multiplies and produces the exotoxin. The infant becomes constipated, listless, generally weak and eats poorly. Death may result from respiratory failure.
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how to determine that the phenotype of a wild type is due to the action of one or two genes?
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classification and characteristics
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