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Where would a predicted silent mutation have to be situated to actually result in a loss-of-function mutation (and potentially lead to the onset of disease)?
A. Intron-exon junction/splice site
B. Translational start site
C. Stop codon
D. Anywhere within the exonic sequences
I have 9 questions in my undergraduate cardiovascular physiology class I need help with. It is mostly dealing with slow response action. I need the work in about 2nite hours from n
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human skeleton
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