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Where would a predicted silent mutation have to be situated to actually result in a loss-of-function mutation (and potentially lead to the onset of disease)?
A. Intron-exon junction/splice site
B. Translational start site
C. Stop codon
D. Anywhere within the exonic sequences
Which is the type of gamete (for a given trait) produced by a dominant homozygous individual? What is the genotypical proportion of these gametes? What about a recessive homozygous
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