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Where would a predicted silent mutation have to be situated to actually result in a loss-of-function mutation (and potentially lead to the onset of disease)?
A. Intron-exon junction/splice site
B. Translational start site
C. Stop codon
D. Anywhere within the exonic sequences
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how biochemical evidence proof DNA as a genetic material?
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PROVIDE a specific example for epithelial and connective tissues, how the arrangement of cells helps with tissue functioning?
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