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Triglyceride accumulation is not a feature of the atherosclerotic plaque but triglyceride-rich lipoproteins also contain cholesterol esters and it is likely that some of these are directly atherogenic.
Hypertriglyceridaemia is associated with alterations in the metabolism of other lipoproteins, which may explain its relationship to CHD risk. It is often inversely related to HDL such that as triglycerides increase, HDL cholesterol concentrations decrease.
In hypertriglyceridaemic individuals there is a preponderance of small, dense LDL particles. A further explanation for the link between plasma triglyceride and CHD risk relates to the association between hypertriglyceridaemia and coagulation factors. Factor VII is an important component of the extrinsic coagulation system and in prospective studies has been shown to be an independent predictor of CHD. Increasing plasma triglycerides are positively correlated with the activity of factor VII and some of the day-to-day variation in factor VII coagulation activity is related to dietary fat intake.
Plasma triglyceride concentration is also positively correlated with activity of plasminogen activator inhibitor 1 (PAI-1). PAI-1 is an inhibitor of plasminogen activation and has been shown to be increased in young myocardial infarction patients.
what is binomial nomenclature? What are the importance of it?
What is the Mode of Nutrition in mosquito?
respiration through moist skin
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Q. Investigations process of acute pericarditis? 1) Blood Examination Erythrocyte sedementaion rate (ESR) may be elevated in tuberculous; collagen and purulent pericarditis.
Are organic solvents like benzene and ether polar or non-polar substances? Benzene and the ethers are molecules without electrically charged portions and therefore they are non
A pair of ovaries are the reproductive organs in woman. They are present just below the kidneys in the abdominal cavity. Each ovary has several sac like structures called ova
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