Why cuatsm was adopted to treatment als mice

Reference no: EM133340544

Question: In the paper "Copper delivery to the CNS by CuATSM effectively treats motor neuron disease in SODG93A mice co-expressing the Copper-Chaperone-for-SOD". Why CuATSM was adopted to treatment ALS mice, rather than free Cu2+? Why CCS gene co-expressing with SOD1 G93A mutant were used to test CuATSM efficacy? What are CuATSM efficacy in this mouse model?

Reference no: EM133340544

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