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Dr. Todd Sacktor, another neuroscientist at SUNY Downstate Medical Center, collaborated with Dr. Fenton to test the prediction that PKMZeta was required to maintain LTM. Researchers postulated that a necessity for LTM was the formation and reinforcement of specific neural connections (brain cells) maintained by the enzyme PKMZeta. Dr. Sacktor established LTM in mice using Dr. Fenton's place avoidance task. Upon learning to avoid the shock zone, mice were administered ZIP, a drug that inhibits PKMzeta. Mice no longer avoided the shock zone.
What was the independent variable? What was the dependent variable? The shock zone was turned off once the mouse was injected with ZIP - should it have been? Explain. Should ZIP be legal administered to individuals that want to erase a memory - why or why not? Give at least two questions that should be answered before administering to humans?
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