Reference no: EM131917317

Biochemists have discovered a protein kinase enzyme named BRAF that is an important link in a molecular pathway that causes a cell to divide. Normally, BRAF responds to signals coming from outside the cell-signals calling for the cell to divide normally under normal conditions. But there is a mutation in BRAF enzymes that causes it activate the cell toward division continually. In this way it gives rise to melanomas and thyroid or ovarian cancers. Biochemists have also found a drug, vemurafenib, which binds selectively to mutant BRAF totally inactivating it. Cells that have inactivated BRAF undergo apoptosis-a process that leads to cell death.

Would this be a reduction of cancer risk, correction of cancer gene, or destruction of cancerous tissue? Looking for the most significant response. I am lending towards correction of cancer gene but am not sure?