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How might the constitutive activation of kras aid

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Reference no: EM132412636

KRAS is a gene commonly found to be mutated in several different types of cancer. It is activated through the dimerization of a receptor tyrosine kinase in response to growth factors, and causes the cell to upregulate glucose intake. KRAS is typically mutated to be constitutively active in cancer patients. 

  1. How might the constitutive activation of KRAS aid in the development of a tumorigenic state in a cell?
  2. What would be the effect on a cell if only one copy of the KRAS protein was mutated? Both copies?
  3. Which of the two classes of cancer-related genes is KRAS? How can you tell?

Reference no: EM132412636

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