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-For patients with BLS 1, their cells may express up to 10% of the MHC class I relative to healthy controls. Although BLS 1 patients may have a homologous non-sense mutation for a TAP-1 or TAP-2 gene, how would you account for the observation that their cells may express up to 10% of the MHC class I proteins relative to healthy controls?
-In patients with suspected BLS II is made, a bone marrow transplant is often considered before a definite diagnosis has been confirmed. Why do you think this is?
-For BLS II patients that undergo successful hematopoietic stem cell transplant no longer have the severe combined immunodeficiency phenotype, and have the antibody responses by B cells improves considerably. However, their total T cell counts are only about 20% of normal. These patients do not have a thymus transplant when they receive the hematopoietic stem cell transplant. How would you explain the partial restoration of their T cells? Why don't they have 100% of the normal T cell count?
1. Identify the stage of respiration being described by each statement:
Which of the following sequences represents the hierarchy of biological organization from the least to the most complexlevel?
What are some differences between eukaryotic cells and prokaryotic cells?
Santo G., a 47 year old Hispanic man with a long history of obesity and type 2 diabetes of recent onset, required daily insulin injections.
UGU is mutated so that the tRNA recognizes the UAG codon but still carries a cysteine residue, how would translation be affected and the resulting proteins?
The bacteriumE. coli is a ‘switch hitter’ – that is, it can grow in the presence or absence of oxygen. However,fermentation is inhibited in the presence of molecular oxygen. Suggest a reason for this observation.
What is the disorder beriberi and how is this relevant to pyruvate dehydrogenase mechanism? and why does its exposure to arsenite or mercury lead to symptons similar to beriberi.
If the gene encoding the enzyme, glycerophosphate acyl tranferase is mutated, how does this affect phospholipid metabolism?
How and where fertilization occurs in lions and if it is internal or external?
LMS is very rare human hereditary disorder resulting in spastic para paraplegia and mental retardation. LMS is caused by a recessive allele with Mendelian inheritance.
While studying the Michaelis - Menton kinetics of enzymes in the TCA cycle, the synthetic hydrocarbon malonate was shown to act as a competitive inhibitor of one of the enzymes.
1 assume the gene for dimples d is recessive. then assume that two individuals with the genotypes dd and dd mate.
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