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Cystic fibrosis is genetic disease caused by mutations in the CFTR gene. The consequence of this is production of an abnormal transmembrane protein that is responsible for producing sweat, mucus, and digestive fluids.
Explain in depth the correlation between the defective gene and the abnormal protein that is produced. Be sure to mention the process involved in protein production, whether or not those process(s) have properly occurred, and their end products.
Describe the 4 types of tissues. Within your discussion, compare and contrast the cells, microscopic characteristics, locations, functions of each tissue type.
Draw the synaptic configuration of homologous pairs of chromosomes when one member is normal and the other member has sustained a duplication within the chromosome
Choose any topic on nutrition and discuss it. Should be two to three sheets long with work cited.
Do you consider barriers to reporting negative results to be a problem? Why or why not? Please provide 2-3 sentences in your own words to justify your answer.
You should consider at least the overall structure, ligand binding, generation of the receptor and secreted forms of the receptor.
This code is what allows the string of nucleotides in our DNA to code for the sequence of amino acids that make up proteins.
What causes the Tay Sach's disease and Cystic fibrosis genetic conditions.
Independent assortment during meiosis deals with Mendel's fourth postulate. It can provide genetic diversity among gametes in meiosis as well as crossing over.
BIOL110 - List the three main types of skeletons found in animals and briefly explain their major advantages or disadvantages
Question 1: How does the cyclisation of fructose occur? can you explain it to me with a diagram with arrows and polarity?
Explain IN DETAIL what behavioral and dietary changes will need to be made by you to make each of the above weaknesses, strengths rather than weaknesses?
Give an example of two missense mutations in the sequence listed here ( AUG CCG AAG UCA UAA). one example that is likely to cause a dramatic change in function, and one that is likely to give a normal phenotype.
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