Reference no: EM132612389

What bacterium pathogen is being described It enter through eyes, mouth, nose, or urogenital openings, or through wounds or bites that breach the skin barrier. It can jeopardize a person's immune system by evading the host through variety of mechanisms. I use my capsule against the phagocytotic action of the host's macrophages and neutrophils. I can release cell components and lipopolysaccharides (LPS) to cause septic shock. It have toxins that can serve to damage the host cells and aid in invasion. adhesins facilitate the binding to host surfaces. Once adhered to a host surface, t may further invade host tissues. Pathogens like this may "burrow" further into a tissue by expressing and secreting proteases and glycanases that digest host extracellular matrix proteins and polysaccharides. In addition, I may also invade the host tissue cells and gain access to the intracellular environment. This can be facilitated by the natural phagocytosis mechanisms of macrophages and neutrophils or by induced uptake where the pathogen signals the host cell to engulf adhered bacteria. This is brought about by my strategy, which is inducing uptake is the use of a type III secretion system which injects bacterial signalling proteins into the host cell. Within the host cell, I will reside within a phagolysome (a phagosome which has fused with a lysosome), a phagosome which has not fused with a lysosome, or within the host cell cytosol. When the host's immune system could not compensate with the damages it caused due to decreasing amount of the white blood cells, which are the main fighters of the immune system, there's a guarantee that I can win this battle.