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Crpv do to change the translation

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Internal initiation of translation, as used by cricket paralysis virus (CrPV), is normally very inefficient compared to cap-dependent translation of cellular mRNAs but ironically the purpose of the virus using this method is to ensure that viral proteins are expressed instead of host proteins-that is, to allow the virus to take over cellular translation. What kinds of things do viruses like CrPV do to change the translation system to ensure that translation of its mRNA is much more efficient than the vast majority of cellular mRNAs?

Reference no: EM132487016

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