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Calculate the Ki for a competitive inhibitor (I) form the following data by Dixon Plot.
Inhibitor Concentration (mM) : 0, 0.5, 1.0, 1.5, 2.0, 3.0;
Velocity (mM/min) in the presence of 5mM substrate: 0.38, 0.26, 0.21, 0.17, 0.14, 0.10;
Velocity (mM/min) in the presence of 10mM substrate: 0.71, 0.59, 0.48, 0.42, 0.36, 0.29;
Explain the term "ecotype" and the significanace relative to revegatation of an are that has been burned?
2-different mutations are identified in a haploid strain of yeast. The first prevents the synthesis of adenine by a nonsense mutation of the ade one gene.
Do you think biochemical testing must be recommended to detect the deleterious gene in Mrs. Lehman. Which one increased their alveolar ventilation, or do they stay the same? Why with appropriate reason.
One way methane is produced is via reduction of carbon dioxide, which serves as electron acceptor, with molecular hydrogen, which serves as electron donor.
As a Nursing student in Advanced Human Anatomy, you were able to observe a total hip replacement. While the O.R. personnel where prepping the patient for the surgical procedure.
When performing the Kirby-Bauer disk diffusion technique what do the microcolonies in the zone of inhibition represent?
Describe precisely what is microbial death? Why does a population of microbes not die instaneously when exposed to an antimicrobial agent
Ecoli with mutated Oric sequences can still replicate its chromosome, though very slowly. One mechanism of this origin independent replication depends on initiation of transcription.
Think about the reaction from the electron transport chain in the mitochondria. Only consider the forward direction the reaction from left to right
Wing coloration in the scarlet tiger moth has previously been shown to behave as a single locus, two allele system with incomplete dominance.
Plants may be 10, 20, 30, 40, 50, 60 or 70 cm high where plant high is under polygenic control. A true-breeding plant that is 10 cm is crossed to another true-breeding plant that is 50 cm.
Both proto-oncogenes and tumor suppressor genes can interact with transcription factors involved cell division. Determine the fundamental difference in how the two function?
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